Welcome to the SWAN Alzheimer Knowledge Base

SWAN is the participatory knowledge base of Alzheimer Disease that YOU can help develop. SWAN is all about how you interpret, debate, ask questions and advance the science.

» FEATURED CONTRIBUTIONS

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» HOT TOPICS (browse all hypotheses)

Changes in presenilin function lead to dementia and neurodegeneration in Alzheimer Disease

Misfolded proteins accumulated into protein aggregates characterizes the pathologic lesions of AD

The molecular mechanisms of neuronal cell death are involved in the dysfunction and death of neurons in AD

Synaptic loss appears to be the most powerful and ubiquitous proximate factor leading to the dementia of AD

Failure of axonal transport might be the underlying basis for neurodegeneration in AD

» MECHANISMS

» HOW TO CONTRIBUTE

BUILD A HYPOTHESIS
CRITIQUE A HYPOTHESIS
NOMINATE A KEY PAPER
HELP FIND CONNECTIONS
PROPOSE NEW FEATURES
ADD SUPPORTING EVIDENCE

» KNOWLEDGE BASE

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SWAN Alzheimer Knowledge Base beta Semantic Web Applications in Neuromedicine

Welcome to the SWAN Alzheimer Knowledge Base

SWAN Alzheimer Knowledge Base beta
Semantic Web Applications in Neuromedicine